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- W2985965304 endingPage "101" @default.
- W2985965304 startingPage "89" @default.
- W2985965304 abstract "Herpes simplex virus (HSV), including HSV-1 and HSV-2, is an important pathogen that can cause many diseases. Usually these diseases are recurrent and incurable. After lytic infection on the surface of peripheral mucosa, HSV can enter sensory neurons and establish latent infection during which viral replication ceases. Moreover, latent virus can re-enter the replication cycle by reactivation and return to peripheral tissues to start recurrent infection. This ability to escape host immune surveillance during latent infection and to spread during reactivation is a viral survival strategy and the fundamental reason why no drug can completely eradicate the virus at present. Although there are many studies on latency and reactivation of HSV, and much progress has been made, many specific mechanisms of the process remain obscure or even controversial due to the complexity of this process and the limitations of research models. This paper reviews the major results of research on HSV latency and reactivation, and discusses future research directions in this field." @default.
- W2985965304 created "2019-11-22" @default.
- W2985965304 creator A5001791914 @default.
- W2985965304 creator A5037329001 @default.
- W2985965304 creator A5065943073 @default.
- W2985965304 date "2019-05-25" @default.
- W2985965304 modified "2023-09-23" @default.
- W2985965304 title "[Mechanisms of herpes simplex virus latency and reactivation]." @default.
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