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- W2991034855 abstract "The glucose-dependent insulinotropic polypeptide, formerly known as gastric inhibitory peptide (GIP) was first isolated in 1973 from porcine small intestine based on its ability to inhibit gastric hydrochloric acid secretion. Soon after, in 1980, it was found that GIP is a weak inhibitor of acid secretion and potent stimulator of insulin post meals. This phenomenon of higher insulin secretion in response to oral glucose as compared to intravenous glucose, at the same plasma glucose level, is called the incretin effect. GIP is considered the most potent incretin hormone, and along with glucagon-like peptide-1 (GLP-1), it contributes to 25 to 70% of the postprandial insulin response." @default.
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- W2991034855 date "2019-08-25" @default.
- W2991034855 modified "2023-09-27" @default.
- W2991034855 title "Physiology, Gastric Inhibitory Peptide" @default.
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