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- W2991340259 abstract "Type I interferons (IFNs) comprise of pro-inflammatory cytokines created, as well as sensed, by all nucleated cells with the main objective of blocking pathogens-driven infections. Owing to this broad range of influence, type I IFNs also exhibit critical functions in many sterile inflammatory diseases and immunopathologies, especially those associated with endoplasmic reticulum (ER) stress-driven signaling pathways. Indeed, over the years accumulating evidence has indicated that the presence of ER stress can influence the production, or sensing of, type I IFNs induced by perturbations like pattern recognition receptor (PRR) agonists, infections (bacterial, viral or parasitic) or autoimmunity. In this article we discuss the link between type I IFNs and ER stress in various diseased contexts. We describe how ER stress regulates type I IFNs production or sensing, or how type I IFNs may induce ER stress, in various circumstances like microbial infections, autoimmunity, diabetes, cancer and other ER stress-related contexts." @default.
- W2991340259 created "2019-12-05" @default.
- W2991340259 creator A5057648493 @default.
- W2991340259 creator A5064707969 @default.
- W2991340259 date "2020-01-01" @default.
- W2991340259 modified "2023-10-05" @default.
- W2991340259 title "Type I interferons and endoplasmic reticulum stress in health and disease" @default.
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