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- W2995338945 abstract "CancerPhysicians who treat bacterial infections and those who treat cancer often face a common challenge: the development of drug resistance. It is well known that when bacteria are exposed to antibiotics, they temporarily increase their mutation rate, thus increasing the chance that a descendant antibiotic-resistant cell will arise. Russo et al. now provide evidence that cancer cells exploit a similar mechanism to ensure their survival after drug exposure (see the Perspective by Gerlinger). They found that human colorectal cancer cells treated with certain targeted therapies display a transient up-regulation of errorprone DNA polymerases and a reduction in their ability to repair DNA damage. Thus, like bacteria, cancer cells can adapt to therapeutic pressure by enhancing their mutability.Science , this issue p. [1473][1]; see also p. [1458][2] [1]: /lookup/doi/10.1126/science.aav4474 [2]: /lookup/doi/10.1126/science.aba1748" @default.
- W2995338945 created "2019-12-26" @default.
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- W2995338945 date "2019-12-19" @default.
- W2995338945 modified "2023-10-18" @default.
- W2995338945 title "A cross-kingdom tale of drug resistance" @default.
- W2995338945 doi "https://doi.org/10.1126/science.366.6472.1467-n" @default.
- W2995338945 hasPublicationYear "2019" @default.
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