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- W3000705061 abstract "Dentoalveolar abscess, a localized accumulation of pus in the alveolar bone, can produce significant changes in alveolar bone proper that may be resulted by dental caries, trauma or pulpitis causing alveolar bone resorption or even loss. Unfortunately, it can extend vertically toward the apex of the tooth. Additionally, a periapical abscess may form in the alveolar bone adjacent to the apex of the tooth. Interestingly, many bacterial products can stimulate osteoclast formation and bone resorption of periapical tissues. This review displayed drugs that can affect bone resorption that classified as (1) Osteoclast inhibitors such as: bisphosphonates, gene therapeutic (Cathepsin K) estrogens, selective estrogen receptor modulators (SERMs), calcitonin monoclonal antibodies such as denosumab and (2) Osteoclast activators such as DMPPYT, nuclear factor-Kappa B (RANK) signaling and adrenomedullin." @default.
- W3000705061 created "2020-01-23" @default.
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- W3000705061 date "2019-12-30" @default.
- W3000705061 modified "2023-10-14" @default.
- W3000705061 title "Current Approaches in Dental Alveolar Abscess in Sight of Bone Related Drugs" @default.
- W3000705061 doi "https://doi.org/10.31487/j.ord.2019.01.05" @default.
- W3000705061 hasPublicationYear "2019" @default.
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