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- W3000903431 abstract "The treatment of Helicobacter pylori usually fails due to their ability to form biofilms and resistance to antibiotics. This might potentially lead to gastric carcinoma and mucosa-associated lymphoid tissue lymphoma. In the present study, we elucidate the potential role of N-acylhomoserine lactonase stabilized silver nanoparticles (AiiA-AgNPs) in treating biofilms produced by H. pylori. AiiA-AgNPs inhibited quorum sensing (QS) by degradation of QS molecules, thereby reducing biofilm formation, urease production, and altering cell surface hydrophobicity of H. pylori. AiiA-AgNPs showed no cytotoxic effects on RAW 264.7 macrophages at the effective concentration (1-5 µM) of antibiofilm activity. In addition, AiiA-AgNP in high concentration (80-100 µM) exhibited cytotoxicity against HCT-15 carcinoma cells, depicting its therapeutic role in treating cancer." @default.
- W3000903431 created "2020-01-30" @default.
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- W3000903431 date "2020-01-21" @default.
- W3000903431 modified "2023-09-27" @default.
- W3000903431 title "Influence of <i>N</i> ‐acylhomoserine lactonase silver nanoparticles on the quorum sensing system of <i>Helicobacter pylori</i> : A potential strategy to combat biofilm formation" @default.
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- W3000903431 doi "https://doi.org/10.1002/jobm.201900537" @default.
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