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- W3003135273 abstract "Extracellular vesicle (EV) trafficking provides for a constitutive mode of cell-cell communication within tissues and between organ systems. Different EV subtypes have been identified that transfer regulatory molecules between cells, influencing gene expression, and altering cellular phenotypes. Evidence from a range of studies suggests that EV trafficking enhances cell survival and resistance to chemotherapy in solid tumors. In acute myeloid leukemia (AML), EVs contribute to the dynamic crosstalk between AML cells, hematopoietic elements and stromal cells and promote adaptation of compartmental bone marrow (BM) function through transport of protein, RNA, and DNA. Careful analysis of leukemia cell EV content and phenotypic outcomes provide evidence that vesicles are implicated in transferring several known key mediators of chemoresistance, including miR-155, IL-8, and BMP2. Here, we review the current understanding of how EVs exert their influence in the AML niche, and identify research opportunities to improve outcomes for relapsed or refractory AML patients." @default.
- W3003135273 created "2020-01-30" @default.
- W3003135273 creator A5005752595 @default.
- W3003135273 creator A5053724309 @default.
- W3003135273 creator A5070745892 @default.
- W3003135273 creator A5079266737 @default.
- W3003135273 date "2020-02-14" @default.
- W3003135273 modified "2023-10-14" @default.
- W3003135273 title "Extracellular Vesicles and Chemotherapy Resistance in the AML Microenvironment" @default.
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- W3003135273 doi "https://doi.org/10.3389/fonc.2020.00090" @default.