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- W3007375730 endingPage "1900290" @default.
- W3007375730 startingPage "1900290" @default.
- W3007375730 abstract "Periodically, a scientific field should examine its early premises. For ubiquitous toxin/antitoxin (TA) systems, several initial paradigms require adjustment based on accumulated data. For example, it is now clear that under physiological conditions, there is little evidence that toxins of TA systems cause cell death and little evidence that TA systems cause persistence. Instead, TA systems are utilized to reduce metabolism during stress, inhibit phages, stabilize genetic elements, and influence biofilm formation (bacterial cells attached via an extracellular matrix). In this essay, it is argued that toxins bound to antitoxins are not likely to become activated by preferential antitoxin degradation but instead, de novo toxin synthesis in the absence of stoichiometric amounts of antitoxin activates toxins." @default.
- W3007375730 created "2020-03-06" @default.
- W3007375730 creator A5006669237 @default.
- W3007375730 creator A5082173877 @default.
- W3007375730 date "2020-02-20" @default.
- W3007375730 modified "2023-10-09" @default.
- W3007375730 title "Toxin/Antitoxin System Paradigms: Toxins Bound to Antitoxins Are Not Likely Activated by Preferential Antitoxin Degradation" @default.
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- W3007375730 doi "https://doi.org/10.1002/adbi.201900290" @default.
- W3007375730 hasPubMedId "https://pubmed.ncbi.nlm.nih.gov/32293143" @default.
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