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- W3008368802 endingPage "357" @default.
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- W3008368802 abstract "Breast cancer (BC) relapse, despite clinical advancement, remains one of the biggest issues in the field. Intercellular communication, specifically via connexin (Cx)-mediated gap junctions (GJs), play a key role in the long-term survival of these, treatment-resistant breast cancer stem cells (CSCs), allowing for relapse. Both basic and clinical evidence reveal dual roles for GJs, in tumor suppression, generally referred to as dormancy, and progression and metastasis. GJ intercellular communication (GJIC) can be mediated by multiple types of Cxs, depending on the organ to which the BC cells metastasize. This review expands on the differential expression of Cx-mediated GJIC between CSCs and niche cells within a given microenvironment." @default.
- W3008368802 created "2020-03-06" @default.
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- W3008368802 date "2020-04-01" @default.
- W3008368802 modified "2023-10-16" @default.
- W3008368802 title "Gap Junctions and Breast Cancer Dormancy" @default.
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- W3008368802 doi "https://doi.org/10.1016/j.trecan.2020.01.013" @default.
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