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- W3008627915 endingPage "104076" @default.
- W3008627915 startingPage "104076" @default.
- W3008627915 abstract "Regulatory B (Breg) cells are characterized by differential expression of CD5 and CD1d in mouse and CD24 and CD38 in human immune systems. The Breg family also includes LAG-3+CD138hi plasma cells, CD1d CD5 CD21 CD23 cells, Tim1, PD-L1, PD-L2, CD200- expressing B cells, and CD39hiKi67+ cells originating from the transitional, marginal zone or germinal centre of the spleen. Breg cells produce IL10 and IL35 and to cause immunosuppression. These cells respond to TLR2, TLR4, and TLR9 agonists, CD40 ligands, IL12p35 and heat shock proteins. Emerging evidence suggests that TLR signalling component Myd88 impacts the modulation of Breg cell responses and the host’s susceptibility to infection. Breg cells are found to reduce relapsing-remitting experimental autoimmune encephalomyelitis. However, the Breg-mediated mechanism used to control T cell-mediated immune responses is still unclear. Here, we review the existing literature to find gaps in the current knowledge and to build a pathway to further research." @default.
- W3008627915 created "2020-03-06" @default.
- W3008627915 creator A5037542629 @default.
- W3008627915 creator A5067621840 @default.
- W3008627915 creator A5088819471 @default.
- W3008627915 date "2020-06-01" @default.
- W3008627915 modified "2023-10-16" @default.
- W3008627915 title "Regulatory B cells in infection, inflammation, and autoimmunity" @default.
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- W3008627915 doi "https://doi.org/10.1016/j.cellimm.2020.104076" @default.
- W3008627915 hasPubMedId "https://pubmed.ncbi.nlm.nih.gov/32143836" @default.
- W3008627915 hasPublicationYear "2020" @default.
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