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- W3008908699 abstract "Background: Retinopathy of prematurity (ROP) is an eye disease of the immature newborn characterized by pathological neovascularization (NV). ROP classically arises due to changes in oxygen availability in the retina. However, other factors, such as red blood cell (RBC) transfusion, independently contribute to disease severity. Heme molecules, rich in RBC, are the primary source of endogenous iron. Heme is metabolized by heme oxygenase (HO) into biliverdin, carbon monoxide, and ferrous ions. Low iron levels stabilize hypoxia-inducible factor 1α (HIF1 α), the main transcription factor of vascular endothelial growth factor (VEGF) that drives angiogenesis. Here we investigate the role of heme metabolism in pathological NV." @default.
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- W3008908699 date "2019-12-01" @default.
- W3008908699 modified "2023-09-27" @default.
- W3008908699 title "AB038. Pathological neovascularization in retinopathy of prematurity is regulated by heme-derived iron trafficking" @default.
- W3008908699 doi "https://doi.org/10.21037/aes.2019.ab038" @default.
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