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- W3010984856 endingPage "100046" @default.
- W3010984856 startingPage "100046" @default.
- W3010984856 abstract "Systemic lupus erythematosus (SLE) is a typical autoimmune disease characterized by chronic inflammation and pathogenic auto-antibodies. Apart from B cells, dysregulation of other immune cells also plays an essential role in the pathogenesis and development of the disease including CD4+T cells, dendritic cells, macrophages and neutrophils. Since metabolic programs control immune cell fate and function, they are critical checkpoints in an effective immune response and are involved in the etiology of autoimmune disease. In addition, mitochondria and oxidative stress are both involved in cellular metabolism and is also essential in immune response. In this review, apart from the disturbed immune system, we will discuss mitochondrial dysfunction, oxidative stress, abnormal metabolism (including glucose, lipid and amino acid metabolism) of immune cells as well as epigenetic control of metabolism reprogramming to elucidate the underlying pathogenic mechanisms of systemic lupus erythematosus." @default.
- W3010984856 created "2020-03-23" @default.
- W3010984856 creator A5019088881 @default.
- W3010984856 creator A5020414704 @default.
- W3010984856 creator A5034551921 @default.
- W3010984856 creator A5041053731 @default.
- W3010984856 creator A5058772771 @default.
- W3010984856 date "2020-01-01" @default.
- W3010984856 modified "2023-10-17" @default.
- W3010984856 title "Immunometabolism in the pathogenesis of systemic lupus erythematosus" @default.
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