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- W3011814882 endingPage "1891" @default.
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- W3011814882 abstract "Traumatic brain injury is known to reprogram the epigenome. Chromatin immunoprecipitation-sequencing of histone H3 lysine 27 acetylation (H3K27ac) and tri-methylation of histone H3 at lysine 4 (H3K4me3) marks was performed to address the transcriptional regulation of candidate regeneration-associated genes. In this study, we identify a novel enhancer region for induced WNT3A transcription during regeneration of injured cortical neurons. We further demonstrated an increased mono-methylation of histone H3 at lysine 4 (H3K4me1) modification at this enhancer concomitant with a topological interaction between sub-regions of this enhancer and with promoter of WNT3A gene. Together, this study reports a novel mechanism for WNT3A gene transcription and reveals a potential therapeutic intervention for neuronal regeneration." @default.
- W3011814882 created "2020-03-23" @default.
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- W3011814882 date "2020-03-10" @default.
- W3011814882 modified "2023-10-10" @default.
- W3011814882 title "Epigenetic Regulation of WNT3A Enhancer during Regeneration of Injured Cortical Neurons" @default.
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- W3011814882 doi "https://doi.org/10.3390/ijms21051891" @default.
- W3011814882 hasPubMedCentralId "https://www.ncbi.nlm.nih.gov/pmc/articles/7084788" @default.
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