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• W3016937018 abstract "Purpose of review To summarize the recent studies directly comparing LDL and lipoprotein(a) as causal factors for cardiovascular disease and mortality. Recent findings In approximately 100,000 individuals from the Copenhagen General Population Study for risk of myocardial infarction, in observational analyses per 39 mg/dl (1 mmol/l) cholesterol increase, the hazard ratio was 1.3 (95% confidence interval: 1.2–1.3) for LDL cholesterol and 1.6 (1.4–1.9) for lipoprotein(a) cholesterol. In corresponding genetic analyses, the causal risk ratio was 2.1 (1.3–3.4) for LDL and 2.0 (1.6–2.6) for lipoprotein(a). Also, a 15 mg/dl (0.39 mmol/l) cholesterol increase was associated with a hazard ratio for cardiovascular mortality of 1.05 (1.04–1.07) for LDL cholesterol and 1.18 (1.12–1.25) for lipoprotein(a) cholesterol. Corresponding values for all-cause mortality were 1.01 (1.00–1.01) for LDL cholesterol and 1.07 (1.04–1.10) for lipoprotein(a) cholesterol. In genetic, causal analyses, the mortality increases for elevated lipoprotein(a) appeared to be through apolipoprotein(a) kringle IV-2 rather than through lipoprotein(a) levels per se. Summary On cholesterol scales, lipoprotein(a) and LDL appeared equal as causal factors for myocardial infarction; however, lipoprotein(a) was most important for mortality. Lipoprotein(a) effects may not only be due to cholesterol content but could also be due to the structure of lipoprotein(a) resembling plasminogen." @default.
• W3016937018 created "2020-04-24" @default.
• W3016937018 creator A5016803860 @default.
• W3016937018 creator A5079728437 @default.
• W3016937018 date "2020-06-01" @default.
• W3016937018 modified "2023-10-05" @default.
• W3016937018 title "Lipoprotein(a)" @default.
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• W3016937018 doi "https://doi.org/10.1097/mol.0000000000000681" @default.
• W3016937018 hasPubMedId "https://pubmed.ncbi.nlm.nih.gov/32304380" @default.
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