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- W3020953627 endingPage "117682" @default.
- W3020953627 startingPage "117682" @default.
- W3020953627 abstract "Atherosclerosis is a disease in which lipid-laden plaques are developed inside the vessel walls of arteries. The immune system is activated, resulting in inflammation and oxidative stress. Endothelial cells (ECs) are activated, arterial smooth muscle cells (SMCs) proliferate, macrophages are activated, and foam cells are developed, leading to dysfunctional ECs. Epigenetic regulatory mechanisms, including DNA methylation, histone modifications, and microRNAs are involved in the modulation of genes that play distinct roles in several aspects of cell biology and physiology, hence linking environmental stimuli to gene regulation. Recent research has investigated the involvement of DNA methylation in the etiopathogenesis of atherosclerosis, and several studies have documented the role of this mechanism in various aspects of the disease. Regulation of DNA methylation plays a critical role in the integrity of ECs, SMC proliferation and formation of atherosclerotic lesions. In this review, we seek to clarify the role of DNA methylation in the development of atherosclerosis through different mechanisms." @default.
- W3020953627 created "2020-05-13" @default.
- W3020953627 creator A5005178143 @default.
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- W3020953627 creator A5046431472 @default.
- W3020953627 creator A5054750006 @default.
- W3020953627 creator A5091337482 @default.
- W3020953627 date "2020-07-01" @default.
- W3020953627 modified "2023-10-05" @default.
- W3020953627 title "Epigenetic control of atherosclerosis via DNA methylation: A new therapeutic target?" @default.
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