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- W3021853018 endingPage "264" @default.
- W3021853018 startingPage "247" @default.
- W3021853018 abstract "Blood coagulation is the process whereby cells and soluble protein elements interact to form an intravascular blood clot. When this occurs in response to vessel injury, it is an important protective mechanism that functions to seal vascular bleeds, thereby prevent excessive hemorrhage. This physiological process is generally referred to as hemostasis. However, in pathological situations, blood coagulation may be triggered by a variety of stimuli and lead to the formation of a maladaptive intravascular clot or thrombus that may obstruct blood flow to or from a critical organ, and/or embolize to a distal site through the circulatory system. This process is known as thrombosis or thromboembolism and it may affect either the arterial or venous circulations. This chapter focuses on particularly defects in the soluble coagulation factors, defects in platelet number or function, other defects leading to hemorrhage, and inherited defects predisposing to thrombosis. Classic hemophilia/hemophilia A (factor VIII deficiency) and Christmas disease/hemophilia B (factor IX deficiency) are the best known examples of the clotting factor deficiencies. The chapter discusses each of these deficiencies in the numerical order ascribed by the Roman numeral classification system." @default.
- W3021853018 created "2020-05-13" @default.
- W3021853018 creator A5013658598 @default.
- W3021853018 creator A5027992566 @default.
- W3021853018 date "2009-01-01" @default.
- W3021853018 modified "2023-09-26" @default.
- W3021853018 title "Molecular Basis of Hemostatic and Thrombotic Diseases" @default.
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- W3021853018 doi "https://doi.org/10.1016/b978-0-12-374419-7.00015-9" @default.
- W3021853018 hasPublicationYear "2009" @default.