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- W3023554034 endingPage "1405" @default.
- W3023554034 startingPage "1385" @default.
- W3023554034 abstract "This chapter discusses metabolic myopathies. Metabolic myopathies sensu stricto are those with defined blocks of metabolic pathways caused by deficient or nonfunctional enzymes. Mitochondrial encephalomyopathies are multisystemic disorders that usually include a metabolic myopathy. Muscle contraction depends on the provision of energy in the form of adenosine triphosphate (ATP). Carbohydrates are the most important source of energy for brief, intense exercise in anaerobic conditions. The glycogenoses are hereditary disorders caused by enzyme deficiencies in glycogen metabolism or glycolysis. They have been assigned Roman numerals in the order of their discovery but are now also specified by the underlying enzyme defect. Muscle damage results from glycogen storage on the one hand and from energy failure on the other. The clinical manifestation of glycogen storage is fixed, progressive, mostly proximal muscle weakness. Many metabolic myopathies discussed in this chapter and several other conditions, especially exposure to therapeutic or recreational drugs, may lead to rhabdomyolysis and myoglobinuria. Even otherwise healthy individuals may have myoglobinuria when they subject themselves to unusually strenuous physical exertion for which they have not been adequately trained. Other causes of myoglobinuria include toxic, ischemic, mechanical, or thermal damage to muscle." @default.
- W3023554034 created "2020-05-13" @default.
- W3023554034 creator A5026565602 @default.
- W3023554034 creator A5026772758 @default.
- W3023554034 date "2003-01-01" @default.
- W3023554034 modified "2023-09-25" @default.
- W3023554034 title "Metabolic Myopathies" @default.
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