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- W3025320105 endingPage "3435" @default.
- W3025320105 startingPage "3435" @default.
- W3025320105 abstract "Cerebral amyloid angiopathy (CAA), one of the main types of cerebral small vessel disease, is a major cause of spontaneous intracerebral haemorrhage and an important contributor to cognitive decline in elderly patients. Despite the number of experimental in vitro studies and animal models, the pathophysiology of CAA is still largely unknown. Although several pathogenic mechanisms including an unbalance between production and clearance of amyloid beta (Aβ) protein as well as ‘the prion hypothesis’ have been invoked as possible disease triggers, they do not explain completely the disease pathogenesis. This incomplete disease knowledge limits the implementation of treatments able to prevent or halt the clinical progression. The continuous increase of CAA patients makes imperative the development of suitable experimental in vitro or animal models to identify disease biomarkers and new pharmacological treatments that could be administered in the early disease stages to prevent irreversible changes and disease progression." @default.
- W3025320105 created "2020-05-21" @default.
- W3025320105 creator A5000596614 @default.
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- W3025320105 date "2020-05-13" @default.
- W3025320105 modified "2023-10-13" @default.
- W3025320105 title "Understanding the Pathophysiology of Cerebral Amyloid Angiopathy" @default.
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