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- W3025675423 endingPage "3506" @default.
- W3025675423 startingPage "3506" @default.
- W3025675423 abstract "Kidney transplantation is the preferred treatment for end-stage kidney disease (ESKD). Compared to maintenance dialysis, kidney transplantation results in improved patient survival and quality of life. Kidneys from living donors perform best; however, many patients with ESKD depend on kidneys from deceased donors. After procurement, donor kidneys are placed in a cold-storage solution until a suitable recipient is located. Sadly, prolonged cold storage times are associated with inferior transplant outcomes; therefore, in most situations when considering donor kidneys, long cold-storage times are avoided. The identification of novel mechanisms of cold-storage-related renal damage will lead to the development of new therapeutic strategies for preserving donor kidneys; to date, these mechanisms remain poorly understood. In this review, we discuss the importance of mitochondrial and proteasome function, protein homeostasis, and renal recovery during stress from cold storage plus transplantation. Additionally, we discuss novel targets for therapeutic intervention to improve renal outcomes." @default.
- W3025675423 created "2020-05-21" @default.
- W3025675423 creator A5018323619 @default.
- W3025675423 creator A5073768179 @default.
- W3025675423 date "2020-05-15" @default.
- W3025675423 modified "2023-10-08" @default.
- W3025675423 title "Targeting Mitochondria during Cold Storage to Maintain Proteasome Function and Improve Renal Outcome after Transplantation" @default.
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