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• W3025806616 endingPage "118733" @default.
• W3025806616 startingPage "118733" @default.
• W3025806616 abstract "Cell responses to reduced oxygen supply (hypoxia) are largely mediated by hypoxia-inducible transcription factors (HIFs). The pathophysiological role of the HIF pathway is driven by its ability to potentiate key biological processes as part of the adaptation to hypoxia, such as erythropoiesis and angiogenesis. Moreover, the role of HIF signaling in the reprogramming of cell metabolism is also critical to understand the role of these transcription factors in health and disease. In this regard, HIFs reprogram oxidative metabolism of glucose and fatty acids, offering a molecular mechanism by which the HIF pathway can help cells become more tolerant of redox stress during hypoxic/ischemic episodes. However, the way in which HIFs influence amino acid metabolism and its pathophysiology consequences have been less well explored. Here we review recent studies about the role of the HIF1α and HIF2α isoforms in amino acid metabolism, which provides insight to better understand how these factors can influence cell autonomous proliferation and cellular tolerance to hypoxia." @default.
• W3025806616 created "2020-05-21" @default.
• W3025806616 creator A5006579573 @default.
• W3025806616 creator A5071826077 @default.
• W3025806616 date "2020-09-01" @default.
• W3025806616 modified "2023-10-02" @default.
• W3025806616 title "Role of the HIF oxygen sensing pathway in cell defense and proliferation through the control of amino acid metabolism" @default.
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• W3025806616 doi "https://doi.org/10.1016/j.bbamcr.2020.118733" @default.
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• W3025806616 hasPublicationYear "2020" @default.
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