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- W3026177532 endingPage "108157" @default.
- W3026177532 startingPage "108157" @default.
- W3026177532 abstract "Smoking remains the leading cause of morbidity and mortality in the United States, with less than 5% of smokers attempting to quit succeeding. This low smoking cessation success rate is thought to be due to the long-term adaptations and alterations in synaptic plasticity that occur following chronic nicotine exposure and withdrawal. Glial cells have recently emerged as active players in the development of dependence phenotypes due to their roles in modulating neuronal functions and synaptic plasticity. Fundamental studies have demonstrated that microglia and astrocytes are crucial for synapse formation and elimination in the developing brain, likely contributing to why glial dysfunction is implicated in numerous neurological and psychiatric disorders. Recently, there is increasing evidence for the involvement of glial cells in drug dependence and its associated behavioral manifestations. This review summarizes the newly evaluated role of microglia and astrocytes as molecular drivers of nicotine dependence and withdrawal phenotypes. This article is part of the special issue on ‘Contemporary Advances in Nicotine Neuropharmacology." @default.
- W3026177532 created "2020-05-29" @default.
- W3026177532 creator A5043322129 @default.
- W3026177532 creator A5054655525 @default.
- W3026177532 creator A5081338293 @default.
- W3026177532 creator A5091032970 @default.
- W3026177532 date "2020-09-01" @default.
- W3026177532 modified "2023-10-11" @default.
- W3026177532 title "Glial cells as therapeutic targets for smoking cessation" @default.
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