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- W302745744 abstract "The cloning of the uncoupling proteins (UCP1) homologues, UCP2 and UCP3, has raised considerable interest in the phenomenon uncoupling. The expression of UCP3 mainly in skeletal muscle mitochondria and the potency of the skeletal muscle as a thermogenic organ made UCP3 an attractive target for studies towards manipulation of energy expenditure to combat pathologies like type 2 diabetes and obesity. Transfecting yeast with UCP3 and overexpressing UCP3 in mice resulted in increased thermogenesis and related desirable phenotypical changes. However, the lack of an apparent phenotype in mice lacking UCP3 triggered the search for alternative functions of UCP3. Data obtained from UCP3-ko mice reveal that UCP3 may be involved in preventing excessive mitochondrial oxidative stress. This chapter reviews UCP3) in relation to its regulation and putative function. In addition, the chapter presents some previously unpublished data indicating that the primary physiological role of UCP3 may not be regulation of energy expenditure." @default.
- W302745744 created "2016-06-24" @default.
- W302745744 creator A5027867322 @default.
- W302745744 creator A5087038295 @default.
- W302745744 date "2003-01-01" @default.
- W302745744 modified "2023-09-24" @default.
- W302745744 title "Physiological significance of uncoupling protein-3: a role in fatty acid handling?" @default.
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- W302745744 doi "https://doi.org/10.1016/s1569-2558(03)33014-0" @default.
- W302745744 hasPublicationYear "2003" @default.
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