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- W3028857100 abstract "Oxidative stress play a key role in the pathogenesis of diabetic peripheral neuropathy. Reactive oxidative species(ROS), derived from hyperglycemia through abnormal mitochondrial electron chain and NADPH oxidase et al, may damage lipids, DNA, and proteins by oxidation. ROS can also promote other classical mechanisms of diabetic peripheral neuropathy, and further cause apoptosis of neurons and Schwann cells, or inflammation of nerve tissue, and finally lead to nerve injury. The relationship between oxidative stress and mechanisms of diabetic peripheral neuropathy was reviewed in this article, in order to get a clue for more effective treatment strategies. (Chin J Endocrinol Metab, 2015, 31: 1000-1003)Key words: Diabetic peripheral neuropathy; Oxidative stress; Reactive oxidative species" @default.
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- W3028857100 date "2015-11-25" @default.
- W3028857100 modified "2023-09-26" @default.
- W3028857100 title "Oxidative stress in diabetic peripheral neuropathy" @default.
- W3028857100 doi "https://doi.org/10.3760/cma.j.issn.1000-6699.2015.11.017" @default.
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