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- W3029032441 abstract "Toll样受体(TLRs)在单核巨噬细胞等免疫细胞中表达,TLRs激活后通过依赖髓样分化因子88(MyD88)和非依赖MyD88信号转导途径,最终引起核因子(NF)-κB活化、炎性反应因子分泌增加.TLRs可能通过增加1型糖尿病遗传易感性、激活病毒感染引起的免疫反应而参与其发病,并且可能在肥胖、2型糖尿病患者的炎性反应、胰岛素抵抗的产生和糖尿病大血管炎性改变中发挥重要作用。" @default.
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- W3029032441 date "2008-03-20" @default.
- W3029032441 modified "2023-09-25" @default.
- W3029032441 title "Toll-like receptors and diabetes" @default.
- W3029032441 doi "https://doi.org/10.3760/cma.j.issn.1673-4157.2008.02.012" @default.
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