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- W3030207907 abstract "嵌合的bcr-abl编码酪氨酸激酶在白血病生成中起决定性的作用.生物学的进步促进了针对CML分子靶向治疗的发展.酪氨酸激酶抑制剂STI571是一个以bcr-abl为靶标的新的治疗药物,最近已被FDA批准为CML的治疗药物,它可以在bcr-abl阳性白血病的患者中诱导血液学和细胞遗传学的缓解,但多种机制导致它有可能发生耐药.如何克服耐药成为血液学家及药物学家致力解决的一个热点问题." @default.
- W3030207907 created "2020-06-05" @default.
- W3030207907 creator A5035763585 @default.
- W3030207907 creator A5054170013 @default.
- W3030207907 date "2003-05-20" @default.
- W3030207907 modified "2023-09-25" @default.
- W3030207907 title "STI571的耐药机制及其克服对策" @default.
- W3030207907 doi "https://doi.org/10.3760/cma.j.issn.1673-419x.2003.03.018" @default.
- W3030207907 hasPublicationYear "2003" @default.
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