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- W3030442908 abstract "BCL-2 family proves to be composed of proapoptotic and antiapoptotic proteins up to now.Antiapoptotic members,such as BCL-2,promote cell survival by inhibiting the function of the proapoptotic BCL-2 proteins such as Bax.Exact mechanism by which BCL-2 family proteins regulates apoptosis is still unclear,but at least three different models of regulation have emerged from the literature.Since the BCL-2 family proteins have the potential to affect multiple mechanisms of apoptosis including calcium dysregulation,and oxidative stess,BCL-2 family members are important regulators of the mitochondrial pathway of apoptosis.These proteins decide whether the mitochondria should initiate the cell death program and release proapoptotic factors such as cytochrome C.It is important for apoptosis to induce pulmonary fibrosis including apoptosis susceptibility in epithelial cells and apoptosis resistance in myofibroblasts.BCL-2 proteins play a key role in regulating apoptosis in the pulmonary fibrosis.This review summarizes the current understanding on role of BCL-2 proteins in the pulmonary fibrosis.Key words: BCL-2; Apoptosis; Pulmonary fibrosis" @default.
- W3030442908 created "2020-06-05" @default.
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- W3030442908 date "2008-12-20" @default.
- W3030442908 modified "2023-09-23" @default.
- W3030442908 title "Relationship of BCL-2 family members.apoptosis with pulmonary fibrosis" @default.
- W3030442908 doi "https://doi.org/10.3760/cma.j.issn.1673-436x.2008.24.007" @default.
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