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- W3032912163 abstract "Abstract Background and Aims Nitric oxide, nitric oxide, nitric oxide or NO is an endogenous molecule released by endothelial cells, macrophages, liver cells and neurons. There are three forms of nitric oxide synthase in the body. : the inducible form (iNO), the neuronal form and the endothelial form (eNO). In this work we are interested in (iNO) in patients with CKD and their impact on cardiac complications. Method A study of 202 patients with chronic renal failure at different stages (2.3.4.5 and 5D) was performed in our department. We investigated the correlation between mean (iNO) values in patients with CKD and its relationship with left ventricular hypertrophy. Results The mean age of the patients is 56.5 ± 17 years with a sex ratio of 1.43. The mean value of (iNO) is 65.82 ± 1.06 μmol / l. Its value increases significantly with the degradation of renal function (p <0.05) (stage 2 (61.75 ± 2.34), stage 3 (63.91 ± 2.21). ), stage 4 (64.08 ± 2.18), stage 5 (67 ± 2.7) and stage 5D (72.47 ± 2.18μmol / l). It is found that the mean value of nitric oxide NO is slightly higher in patients with HVG + ( 67.58 ± 1.48) versus HVG-patients (63.1 ± 1.39) (p <0.05).Discussion: iNOS secreted nitric oxide (NO) initiates cardiac remodeling to lead to left ventricular hypertrophy and cardiac fibrosis Conclusion Cardiac complications are the leading cause of death in patients with chronic renal failure. oxidative stress is a risk factor for these cardiac complications, iNOS accelerates cardiac aging." @default.
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- W3032912163 date "2020-06-01" @default.
- W3032912163 modified "2023-09-27" @default.
- W3032912163 title "P0953NITRIC OXIDE AND LEFT VENTRICULAR HYPERTROPHY IN PATIENTS WITH CHRONIC RENAL FAILURE" @default.
- W3032912163 doi "https://doi.org/10.1093/ndt/gfaa142.p0953" @default.
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