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- W3036006926 abstract "ABSTRACT Influenza A viruses cause a spectrum of responses, from mild cold-like symptoms to severe respiratory illness and death. Viral strains and intrinsic host factors, such as age, can influence the severity of the disease. Glycosylation plays a critical role in influenza pathogenesis, however the molecular drivers of influenza outcomes remain unknown. In this work, we characterized the glycomic response to the H1N1 2009 pandemic influenza A virus in age-dependent severity. Using a ferret model and a lectin microarray technology we have developed, we compared responses in newly weaned and aged animals, a model for young children and the elderly, respectively. Glycomic analysis revealed changes in glycosylation over the course of the infection, that were associated with severity in an age-dependent manner. These responses may help explain the differential susceptibility to influenza A virus infection of young children and the elderly." @default.
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- W3036006926 date "2020-06-22" @default.
- W3036006926 modified "2023-09-23" @default.
- W3036006926 title "Age-dependent glycomic response to the 2009 pandemic H1N1 influenza virus and its association with disease severity" @default.
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- W3036006926 doi "https://doi.org/10.1101/2020.06.22.165613" @default.
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