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• W3036402806 endingPage "030006052093160" @default.
• W3036402806 startingPage "030006052093160" @default.
• W3036402806 abstract "Osteoarthritis (OA) is a chronic joint disorder that causes degeneration of cartilage, synovial inflammation, and formation of osteophytes. Aging, obesity, and sex are considered the main risk factors of OA. Recent studies have suggested that metabolic syndrome (MetS) disorders, such as hypertension, hyperlipidemia, diabetes mellitus, and obesity, may be involved in the pathogenesis and progression of OA. MetS disorders are common diseases that also result in atherosclerosis. Researchers believe that OA and atherosclerosis have underlying similar molecular mechanisms because the prevalence of both diseases increases with age. Oxidation of low-density lipoprotein (ox-LDL) is believed to play a role in the pathogenesis of atherosclerosis. Recent reports have shown that ox-LDL and low-density lipoprotein receptor 1 (LOX-1) are involved in the pathogenesis of OA. The purpose of this narrative review is to summarize the current understanding of the role of the LOX-1/ox-LDL system in the pathogenesis of OA and to reveal common underlying molecular pathways that are shared by MetS in OA and the LOX-1/ox-LDL system." @default.
• W3036402806 created "2020-06-25" @default.
• W3036402806 creator A5042511869 @default.
• W3036402806 creator A5081576265 @default.
• W3036402806 date "2020-06-01" @default.
• W3036402806 modified "2023-10-14" @default.
• W3036402806 title "The role of oxidation of low-density lipids in pathogenesis of osteoarthritis: A narrative review" @default.
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• W3036402806 doi "https://doi.org/10.1177/0300060520931609" @default.
• W3036402806 hasPubMedCentralId "https://www.ncbi.nlm.nih.gov/pmc/articles/7303502" @default.
• W3036402806 hasPubMedId "https://pubmed.ncbi.nlm.nih.gov/32552129" @default.
• W3036402806 hasPublicationYear "2020" @default.
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