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- W3036693919 abstract "Substantial evidence suggests that the increase in cytosolic Ca 2+ which occurs upon stimulation of cardiac muscle originates from the sarcoplasmic reticulum (SR) and that its release is triggered by a transient rise in cytosolic Ca 2+ . The junctional SR has been proposed as a Ca 2+ release site, based on a number of observations, e.g. measurements by EPMA have demonstrated significant Ca 2+ stores in the junctional SR in resting cardiac muscle; the ryanodine-sensitive Ca 2+ release channels are associated with junctional SR membrane and the low affinity Ca 2+ binding protein, calsequestrin, is localized to the junctional SR. Although considerable evidence implicates the junctional SR as an intracellular Ca 2+ store in cardiac muscle, release of Ca 2+ from the junctional SR, upon muscle stimulation, has not been directly demonstrated. We therefore propose to use EPMA to directly measure the Ca 2+ content of junctional SR in both relaxed and contracted cardiac muscle to determine whether Ca 2+ is released from the junctional SR during contraction." @default.
- W3036693919 created "2020-06-25" @default.
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- W3036693919 date "1990-08-12" @default.
- W3036693919 modified "2023-09-27" @default.
- W3036693919 title "Subcellular Calcium (Ca2+) Redistribution During Cardiac Muscle Contraction by Electron Probe Microanalysis (EPMA)" @default.
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- W3036693919 doi "https://doi.org/10.1017/s0424820100134272" @default.
- W3036693919 hasPublicationYear "1990" @default.
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