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- W3036698027 endingPage "334" @default.
- W3036698027 startingPage "313" @default.
- W3036698027 abstract "Fungal infections with increasing resistance to conventional therapies are a growing concern. Candida albicans is a major opportunistic yeast responsible for mucosal and invasive infections. Targeting the initial step of the infection process (i.e., C. albicans adhesion to the host cell) is a promising strategy. A wide variety of molecules can interfere with adhesion processes via an assortment of mechanisms. Herein, we focus on how small molecules disrupt biosynthesis of fungal cell wall components and membrane structure, prevent the localization of GPI-anchor proteins, inhibit production of enzymes involved in adhesion, downregulate genes encoding adhesins and competitively inhibit receptor interactions. As a result, adhesion of C. albicans to host cells is reduced, paving the way to new classes of antifungal agents." @default.
- W3036698027 created "2020-06-25" @default.
- W3036698027 creator A5009702512 @default.
- W3036698027 creator A5011612359 @default.
- W3036698027 creator A5028235761 @default.
- W3036698027 date "2021-02-01" @default.
- W3036698027 modified "2023-10-07" @default.
- W3036698027 title "Targeting adhesion in fungal pathogen <i>Candida albicans</i>" @default.
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