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- W3037771224 endingPage "534" @default.
- W3037771224 startingPage "521" @default.
- W3037771224 abstract "Abstract Alcohol consumption is increasing worldwide. Many child‐bearing‐aged women consume alcohol during pregnancy, intentionally or unintentionally, thereby increasing the potential risk for severe congenital diseases. Congenital heart disease (CHD) is the most common birth defect worldwide and can result from both hereditary and acquired factors. Prenatal alcohol exposure (PAE) is considered a key factor that leads to teratogenesis in CHD and its specific phenotypes, especially defects of the cardiac septa, cardiac valves, cardiac canals, and great arteries, adjacent to the chambers, both in animal experiments and clinical retrospective studies. The mechanisms underlying CHD and its phenotypes caused by PAE are associated with changes in retinoic acid biosynthesis and its signaling pathway, apoptosis and defective function of cardiac neural crest cells, disturbance of the Wntβ‐catenin signaling pathway, suppression of bone morphogenetic protein (BMP) signaling, and other epigenetic mechanisms. Drug supplements and early diagnosis can help prevent PAE from inducing CHDs." @default.
- W3037771224 created "2020-07-02" @default.
- W3037771224 creator A5002929086 @default.
- W3037771224 creator A5009674897 @default.
- W3037771224 creator A5020177316 @default.
- W3037771224 creator A5042204958 @default.
- W3037771224 creator A5053726259 @default.
- W3037771224 date "2020-06-24" @default.
- W3037771224 modified "2023-10-14" @default.
- W3037771224 title "Prenatal alcohol exposure induced congenital heart diseases: From bench to bedside" @default.
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