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• W3037819162 startingPage "030006052093210" @default.
• W3037819162 abstract "Azithromycin (AZM) has been used to treat chronic inflammatory airway diseases because it regulates cell–cell contact between airway epithelial cells. Airway mucus hypersecretion is an important component of chronic respiratory diseases. Mucin 5AC (MUC5AC) is the major mucin produced by airway epithelial cells, and hypersecretion of MUC5AC is a sign of various pulmonary inflammatory diseases. Recently, it was found that matrix metallopeptidase 9 is involved in mucus hypersecretion. Moreover, AZM can inhibit the ability of TNF-α-to induce interleukin (IL)-8 production. This review focuses on the effects on AZM that may be beneficial in inhibiting MUC5AC, matrix metalloprotease-9 and IL-8 production in airway epithelial cells. In addition, recent studies have begun to assess activation of mitogen-activated protein kinase (MAPK) signaling pathways in response to AZM. Understanding these new developments may be helpful for clinicians." @default.
• W3037819162 created "2020-07-02" @default.
• W3037819162 creator A5088695098 @default.
• W3037819162 date "2020-06-01" @default.
• W3037819162 modified "2023-10-17" @default.
• W3037819162 title "Mechanism of azithromycin in airway diseases" @default.
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• W3037819162 doi "https://doi.org/10.1177/0300060520932104" @default.
• W3037819162 hasPubMedCentralId "https://www.ncbi.nlm.nih.gov/pmc/articles/7323306" @default.
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