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- W3039166714 endingPage "726" @default.
- W3039166714 startingPage "726" @default.
- W3039166714 abstract "In diseases where epigenetic mechanisms are changed, such as cancer, many genes show altered gene expression and inhibited genes become activated. Human endogenous retrovirus type K (HERV-K) expression is usually inhibited in normal cells from healthy adults. In tumor cells, however, HERV-K mRNA expression has been frequently documented to increase. Importantly, HERV-K-derived proteins can act as tumor-specific antigens, a class of neoantigens, and induce immune responses in different types of cancer. In this review, we describe the function of the HERV-K HML-2 subtype in carcinogenesis as biomarkers, and their potential as targets for cancer immunotherapy." @default.
- W3039166714 created "2020-07-10" @default.
- W3039166714 creator A5000173153 @default.
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- W3039166714 creator A5071137953 @default.
- W3039166714 creator A5075587053 @default.
- W3039166714 creator A5088644610 @default.
- W3039166714 date "2020-07-06" @default.
- W3039166714 modified "2023-10-16" @default.
- W3039166714 title "Human Endogenous Retrovirus K in Cancer: A Potential Biomarker and Immunotherapeutic Target" @default.
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