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- W3039592849 endingPage "1046" @default.
- W3039592849 startingPage "1034" @default.
- W3039592849 abstract "Inflammatory bowel disease (IBD) is associated with the production of reactive species that target cysteine redox switches in proteins, thereby affecting gene regulation, DNA damage, ion transport, intermediary metabolism, and mitochondrial function. Precursors of reactive species are derived from organic and inorganic compounds and their cofactors, including amino acids, vitamins, oxygen, nitrite, and sulfate. Nutrition and the gut microbiome fuel this process to a significant extent. The production of reactive species in IBD is reflected by a reduction in systemic free thiols, the major components of the antioxidant machinery. Systemic free thiols are amenable to nutritional or therapeutic intervention. This opens up future avenues for therapeutic modulation of redox status in IBD." @default.
- W3039592849 created "2020-07-10" @default.
- W3039592849 creator A5001756705 @default.
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- W3039592849 creator A5036280404 @default.
- W3039592849 creator A5068944242 @default.
- W3039592849 date "2020-11-01" @default.
- W3039592849 modified "2023-10-14" @default.
- W3039592849 title "Oxidative Stress and Redox-Modulating Therapeutics in Inflammatory Bowel Disease" @default.
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- W3039592849 doi "https://doi.org/10.1016/j.molmed.2020.06.006" @default.