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• W3040386039 endingPage "94" @default.
• W3040386039 startingPage "85" @default.
• W3040386039 abstract "Variations of synaptic strength are thought to underlie forms of learning and can functionally reshape neural circuits. Metabotropic glutamate receptors play key roles in regulating the strength of chemical synapses. However, information within neural circuits is also conveyed via a second modality of transmission: gap junction-mediated synapses. We review here evidence indicating that metabotropic glutamate receptors also play important roles in the regulation of synaptic communication mediated by neuronal gap junctions, also known as 'electrical synapses'. Activity-driven interactions between metabotropic glutamate receptors and neuronal gap junctions can lead to long-term changes in the strength of electrical synapses. Further, the regulatory action of metabotropic glutamate receptors on neuronal gap junctions is not restricted to adulthood but is also of critical relevance during brain development and contributes to the pathological mechanisms that follow brain injury." @default.
• W3040386039 created "2020-07-10" @default.
• W3040386039 creator A5015110091 @default.
• W3040386039 creator A5053701037 @default.
• W3040386039 date "2021-02-01" @default.
• W3040386039 modified "2023-10-14" @default.
• W3040386039 title "Regulatory Roles of Metabotropic Glutamate Receptors on Synaptic Communication Mediated by Gap Junctions" @default.
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• W3040386039 doi "https://doi.org/10.1016/j.neuroscience.2020.06.034" @default.
• W3040386039 hasPubMedCentralId "https://www.ncbi.nlm.nih.gov/pmc/articles/7805574" @default.
• W3040386039 hasPubMedId "https://pubmed.ncbi.nlm.nih.gov/32619474" @default.
• W3040386039 hasPublicationYear "2021" @default.
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