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- W3040768674 abstract "Liver fibrosis is an intrinsic repair process of chronic injury with excessive deposition of extracellular matrix. As an early stage of various liver diseases, liver fibrosis is a reversible pathological process. Therefore, if not being controlled in time, liver fibrosis will evolve into cirrhosis, liver failure, and liver cancer. It has been demonstrated that hepatic stellate cells (HSCs) play a crucial role in the formation of liver fibrosis. In particular, the activation of HSCs is a key step for liver fibrosis. Recent researches have suggested that autophagy and inflammasome have biological effect on HSC activation. Herein, we review current studies about the impact of autophagy and NOD-like receptors containing pyrin domain 3 (NLRP3) inflammasome on liver fibrosis and the underlying mechanisms." @default.
- W3040768674 created "2020-07-16" @default.
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- W3040768674 date "2020-07-11" @default.
- W3040768674 modified "2023-10-09" @default.
- W3040768674 title "The Role of Autophagy and NLRP3 Inflammasome in Liver Fibrosis" @default.
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- W3040768674 doi "https://doi.org/10.1155/2020/7269150" @default.
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