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- W3042309757 endingPage "218" @default.
- W3042309757 startingPage "191" @default.
- W3042309757 abstract "Neutrophils are critical to innate immunity, including host defense against bacterial and fungal infections. They achieve their host defense role by phagocytosing pathogens, secreting their granules full of cytotoxic enzymes, or expelling neutrophil extracellular traps (NETs) during the process of NETosis. NETs are weblike DNA structures decorated with histones and antimicrobial proteins released by activated neutrophils. Initially described as a means for neutrophils to neutralize pathogens, NET release also occurs in sterile inflammation, promotes thrombosis, and can mediate tissue damage. To effectively manipulate this double-edged sword to fight a particular disease, researchers must work toward understanding the mechanisms driving NETosis. Such understanding would allow the generation of new drugs to promote or prevent NETosis as needed. While knowledge regarding the (patho)physiological roles of NETosis is accumulating, little is known about the cellular and biophysical bases of this process. In this review, we describe and discuss our current knowledge of the molecular, cellular, and biophysical mechanisms mediating NET release as well as open questions in the field." @default.
- W3042309757 created "2020-07-23" @default.
- W3042309757 creator A5032584210 @default.
- W3042309757 creator A5066880935 @default.
- W3042309757 creator A5076909940 @default.
- W3042309757 creator A5077476585 @default.
- W3042309757 date "2020-10-06" @default.
- W3042309757 modified "2023-10-14" @default.
- W3042309757 title "Cellular Mechanisms of NETosis" @default.
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