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• W3044087890 abstract "Abstract To reveal the impacts of smoking on genetic architecture of human body weight, we conducted a genome-wide association study on 5,336 subjects in four ethnic populations from MESA (The Multi-Ethnic Study of Atherosclerosis) data. A full genetic model was applied to association mapping for analyzing genetic effects of additive, dominance, epistasis, and their ethnicity-specific effects. Both the unconditional model (base) and conditional model including smoking as a cofactor were investigated. There were 10 SNPs involved in 96 significant genetic effects detected by the base model, which accounted for a high heritability (61.78%). Gene ontology analysis revealed that a number of genetic factors are related to the metabolic pathway of benzopyrene, a main compound in cigarettes. Smoking may play important roles in genetic effects of dominance, dominance-related epistasis, and gene-ethnicity interactions on human body weight. Gene effect prediction shows that the genetic effects of smoking cessation on body weight vary from different populations." @default.
• W3044087890 created "2020-07-29" @default.
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• W3044087890 date "2020-07-22" @default.
• W3044087890 modified "2023-10-07" @default.
• W3044087890 title "Conditional and unconditional genome-wide association study reveal complicate genetic architecture of human body weight and impacts of smoking" @default.
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• W3044087890 doi "https://doi.org/10.1038/s41598-020-68935-x" @default.
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