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- W3044657543 abstract "Hemiplegic migraine (HM) is a rare migraine subtype with aura presenting as recurrent transient attacks of migraine and hemiparesis. HM is genetically and clinically heterogeneous with underlying heterozygous mutations of genes encoding voltage-gated ion channels for calcium (CACNA1A) and sodium (SCN1A), the isoform 2 of Na+-K+-ATPase’s α-subunit (ATP1A2), as well as transmembrane proteins (PRRT2). 1 Dreier J.P. Victorov I.V. Petzold G.C. et al. Electrochemical failure of the brain cortex is more deleterious when it is accompanied by low perfusion. Stroke. 2013; 44: 490-496 Crossref PubMed Scopus (25) Google Scholar Typically, motor symptoms of migraine auras last less than 72 hours; in some patients muscle weakness may last for longer periods and even result in permanent neurological deficit. 2 Ho M.-L. Rojas R. Eisenberg R.L. Cerebral edema. AJR Am J Roentgenol. 2012; 199: 258-273 Crossref PubMed Scopus (57) Google Scholar No evidence-based guidelines for the emergency management of HM have been established, and management therefore remains empirical. We report the successful treatment of a prolonged ATP1A2 gene-linked HM attack with nimodipine, which has not been reported so far and may be a treatment option for further patients with HM." @default.
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- W3044657543 date "2020-11-01" @default.
- W3044657543 modified "2023-10-06" @default.
- W3044657543 title "Intravenous Nimodipine Treatment for Severe Episode of ATP1A2 Hemiplegic Migraine" @default.
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- W3044657543 doi "https://doi.org/10.1016/j.pediatrneurol.2020.07.009" @default.
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