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- W3048150263 abstract "Abstract The nuclear factor-κB (NF-κB) signaling pathway regulates multiple processes in innate and adaptive immune cells. This pathway is involved in inflammation through the regulation of cytokines, chemokines, and adhesion molecules expression. The NF-κB transcription factor also participates in the survival, proliferation, and differentiation of cells. Therefore, deregulated NF-κB activation contributes to the pathogenesis of inflammatory diseases. Rheumatoid arthritis (RA) is classified as a heterogeneous and complex autoimmune inflammatory disease. Although different immune and non-immune cells contribute to the RA pathogenesis, fibroblast-like synoviocytes (FLSs) play a crucial role in disease progression. These cells are altered during the disease and produce inflammatory mediators, including inflammatory cytokines and matrix metalloproteinases, which result in joint and cartilage erosion. Among different cell signaling pathways, it seems that deregulated NF-κB activation is associated with the inflammatory picture of RA. NF-κB activation can also promote the proliferation of RA-FLSs as well as the inhibition of FLS apoptosis that results in hyperplasia in RA synovium. In this review, the role of NF-κB transcription factor in immune and non-immune cells (especially FLSs) that are involved in RA pathogenesis are discussed." @default.
- W3048150263 created "2020-08-13" @default.
- W3048150263 creator A5003453936 @default.
- W3048150263 creator A5026969651 @default.
- W3048150263 creator A5027436901 @default.
- W3048150263 creator A5056940060 @default.
- W3048150263 creator A5056946140 @default.
- W3048150263 creator A5063085521 @default.
- W3048150263 date "2020-08-08" @default.
- W3048150263 modified "2023-10-18" @default.
- W3048150263 title "NF-κB signaling in rheumatoid arthritis with focus on fibroblast-like synoviocytes" @default.
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