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- W3048334606 endingPage "11" @default.
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- W3048334606 abstract "Ischemic injury triggers a heightened inflammatory response that is essential for tissue repair, but excessive and chronic inflammatory responses contribute to the pathogenesis of ischemic cardiovascular disease. Regulatory T cells (Tregs), a major regulator of self-tolerance and immune suppression, control innate and adaptive immune responses, modulate specific immune cell subsets, prevent excessive inflammation, and participate in tissue repair after ischemia. Herein, we summarize the multiple potential mechanisms by which Tregs exert suppressor functions including modulation of cytokine production, alteration of cell-cell interactions, and disruption of metabolic pathways. Furthermore, we review the role of Tregs implicated in ischemic injury and repair including myocardial, limb, and cerebral ischemia. We conclude with a perspective on the therapeutic opportunities and future challenges of Treg biology in understanding the pathogenesis of ischemic cardiovascular disease states." @default.
- W3048334606 created "2020-08-13" @default.
- W3048334606 creator A5024294938 @default.
- W3048334606 creator A5045516967 @default.
- W3048334606 date "2020-10-01" @default.
- W3048334606 modified "2023-10-16" @default.
- W3048334606 title "Regulatory T cells in ischemic cardiovascular injury and repair" @default.
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