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- W3048409567 endingPage "469" @default.
- W3048409567 startingPage "455" @default.
- W3048409567 abstract "Accumulating evidence suggests that hepatitis B virus (HBV) biological features may influence the course and clinical manifestations of infection and possibly the development of fulminant hepatitis (FH). Since HBV is not a cytocidal virus, virus-induced liver damage results from an interplay between the virus replication and the host's defense. Therefore, viral factors contributing to enhanced replication, induction of a stronger immune attack or apoptosis of hepatocytes could be crucial in development of FH. Numerous mutations in basal core promoter, pre-C, C and S regions of the HBV genome contribute to development of FH by different mechanisms, including enhanced viral replication, the loss of a decoy for immune response, unbalanced expression of viral proteins and retention of unprocessed cytotoxic proteins in hepatocytes." @default.
- W3048409567 created "2020-08-18" @default.
- W3048409567 creator A5027045298 @default.
- W3048409567 creator A5051206346 @default.
- W3048409567 creator A5070337563 @default.
- W3048409567 creator A5080087240 @default.
- W3048409567 date "2020-07-01" @default.
- W3048409567 modified "2023-10-17" @default.
- W3048409567 title "Biological features of hepatitis B virus strains associated with fulminant hepatitis" @default.
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