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- W3048651332 endingPage "960" @default.
- W3048651332 startingPage "949" @default.
- W3048651332 abstract "Numerous experimental and postmortem studies have increasingly reported dystrophic axons and dendrites, and alterations of dendritic spine morphology and density in the hippocampus as prominent changes in the early stages of Alzheimer’s disease (AD). Furthermore, these alterations tend to correlate well with the progressive cognitive decline observed in AD. For these reasons, and because these neurite structures have a capacity to re-grow, re-establish lost connections, and are critical for learning and memory, there is compelling evidence to suggest that therapeutic interventions aimed at preventing their degradation or promoting their regrowth may hold tremendous promise in preventing the progression of AD. In this regard, collapsin response mediator proteins (CRMPs), a family of phosphoproteins playing a major role in axon guidance and dendritic growth, are especially interesting. The roles these proteins play in neurons and immune cells are reviewed here." @default.
- W3048651332 created "2020-08-18" @default.
- W3048651332 creator A5012031138 @default.
- W3048651332 creator A5013926479 @default.
- W3048651332 creator A5041777589 @default.
- W3048651332 creator A5056060375 @default.
- W3048651332 creator A5060358830 @default.
- W3048651332 creator A5078663344 @default.
- W3048651332 date "2020-09-29" @default.
- W3048651332 modified "2023-10-04" @default.
- W3048651332 title "Collapsin Response Mediator Proteins: Novel Targets for Alzheimer’s Disease" @default.
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