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- W3048656673 endingPage "35" @default.
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- W3048656673 abstract "Non-communicable diseases (NCDs) that involve neurodegenerative disorders and metabolic disease impact over 400 million individuals globally. Interestingly, metabolic disorders, such as diabetes mellitus, are significant risk factors for the development of neurodegenerative diseases. Given that current therapies for these NCDs address symptomatic care, new avenues of discovery are required to offer treatments that affect disease progression. Innovative strategies that fill this void involve the mechanistic target of rapamycin (mTOR) and its associated pathways of mTOR complex 1 (mTORC1), mTOR complex 2 (mTORC2), AMP activated protein kinase (AMPK), trophic factors that include erythropoietin (EPO), and the programmed cell death pathways of autophagy and apoptosis. These pathways are intriguing in their potential to provide effective care for metabolic and neurodegenerative disorders. Yet, future work is necessary to fully comprehend the entire breadth of the mTOR pathways that can effectively and safely translate treatments to clinical medicine without the development of unexpected clinical disabilities." @default.
- W3048656673 created "2020-08-18" @default.
- W3048656673 creator A5000415542 @default.
- W3048656673 date "2020-01-01" @default.
- W3048656673 modified "2023-10-05" @default.
- W3048656673 title "Dysregulation of metabolic flexibility: The impact of mTOR on autophagy in neurodegenerative disease" @default.
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