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• W3048785826 abstract "Background: Benign prostatic hyperplasia (BPH) is the most common urologic disease affecting aging men. The pathogenesis of BPH is multi-factorial, and chronic inflammation (CI) might be the central mechanism. Interleukin (IL)-27 signaling has been suggested as a modulator in autoimmune and inflammatory conditions. In this study, we used microarray experiments to analyze gene expression and molecular phenotypic associated with BPH progression, with a particular focus on CI and IL-27/IL-27RA signaling, and verified the microarray data in cell biology experiments." @default.
• W3048785826 created "2020-08-18" @default.
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• W3048785826 date "2020-08-01" @default.
• W3048785826 modified "2023-10-16" @default.
• W3048785826 title "IL-27/IL-27RA signaling may modulate inflammation and progression of benign prostatic hyperplasia via suppressing the LPS/TLR4 pathway" @default.
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• W3048785826 doi "https://doi.org/10.21037/tcr-20-1509" @default.
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