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- W3048832907 abstract "The adrenocortical dysplasia (acd) mouse mutation arose spontaneously in the dwarf strain in 1985 at the Jackson Laboratory. Both the background strain and the acd/acd mice are agouti/leaden. Mice homozygous for the adrenocortical dysplasia mutation (acd/acd) are identifiable at birth by reduced body weight and a high incidence of polydactyly of the hind feet and kinking of the tail. Examination of plucked hairs revealed the presence of dystrophic overhairs (awls and auchene hairs), but an absence of zigzag underhairs. Adrenal cortical morphology and organization were dramatically altered in the (acd/acd) mice compared to littermate controls, hence the designation of this new mutation. Adrenocortical dysplasia is due to a single autosomal recessive mutation. Hypoadrenocorticism (chronic adrenocortical insufficiency) has been recognized as a distinct, yet not uncommon disease in the dog. Dogs with these diseases develop hyperpigmentation of the skin, particularly over points of friction and wear." @default.
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- W3048832907 date "2020-08-26" @default.
- W3048832907 modified "2023-10-17" @default.
- W3048832907 title "The Adrenocortical Dysplasia (acd) Mutation, Chromosome 8" @default.
- W3048832907 doi "https://doi.org/10.1201/9781003068952-18" @default.
- W3048832907 hasPublicationYear "2020" @default.
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