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- W30538813 abstract "Adherence of Plasmodium falciparum-infected erythrocytes (PRBCs) to the microvascular endothelium of specific organs and consequent sequestration is believed to be responsible for the development of malaria pathology. A number of studies have shown that cell adhesion molecules expressed on the surface of endothelial cells mediate the adherence. Recent studies indicate that a subpopulation of PRBCs adhere to chondroitin 4-sulfate (C4S). This adhesion can be effectively inhibited by C4S oligosaccharides. In pregnant women, the placenta specifically selects C4S-adherent PRBCs, and thus these phenotypes multiply and sequester in the intervillous spaces. Over successive pregnancies, women develop a protective humoral response to the C4S-adhesion phenotype. Disruption of C4S-mediated PRBCs adhesion using either a C4S oligosaccharide mimetic or an antiC4S-adhesion vaccine can be an efficient strategy for the treatment of malaria caused by C4S-adherent P. falciparum." @default.
- W30538813 created "2016-06-24" @default.
- W30538813 creator A5059907819 @default.
- W30538813 creator A5067839823 @default.
- W30538813 date "1999-08-01" @default.
- W30538813 modified "2023-09-25" @default.
- W30538813 title "Adherence of Plasmodium falciparum-Infected Erythrocytes to Chondroitin 4-Sulfate" @default.
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- W30538813 doi "https://doi.org/10.1023/a:1020542206916" @default.
- W30538813 hasPubMedId "https://pubmed.ncbi.nlm.nih.gov/10589991" @default.
- W30538813 hasPublicationYear "1999" @default.
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