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- W3068908047 abstract "Ferroptosis was recently identified as an iron-dependent regulatory necrosis process mediated by polyunsaturated fatty acid (PUFA) peroxidation. The pivotal events related to oxidative stress in ferroptosis include direct or indirect glutathione peroxidase 4 (GPX4) inhibition, ferrous iron overload, and lipid peroxidation. The links between ferroptosis and multiple pathological processes including tumor and cardiovascular system disease have become increasingly apparent, and the mechanisms and compounds involved in ferroptosis, such as reduction of coenzyme Q 10 (ubiquinone/CoQ 10 ), are gradually emerging. Current reports have revealed crossroads between ferroptosis and other multiple responses. This overview of the current research illuminates the mechanisms involving ferroptosis-related compounds and emphasizes the crosstalk between ferroptosis and other responses, including mitochondrial damage, endoplasmic reticulum stress (ER stress), autophagy, and the release of damage-associated molecular patterns (DAMPs), to reveal the intersections of regulatory mechanisms. This review also outlines the discovery, characterization, and pathological relevance of ferroptosis and notes controversial elements in ferroptosis-related mechanisms, such as nuclear factor E2-related factor 2 (Nrf2), sequestosome 1 (p62/SQSTM1), and heat shock protein family A member 5 (HSPA5). We hope our inferences will supply a partial reference for disorder prevention and treatment." @default.
- W3068908047 created "2020-08-24" @default.
- W3068908047 creator A5005522695 @default.
- W3068908047 creator A5006906014 @default.
- W3068908047 creator A5012690842 @default.
- W3068908047 creator A5026950458 @default.
- W3068908047 creator A5063237870 @default.
- W3068908047 creator A5080723807 @default.
- W3068908047 date "2020-08-18" @default.
- W3068908047 modified "2023-10-15" @default.
- W3068908047 title "Revisiting Tumors and the Cardiovascular System: Mechanistic Intersections and Divergences in Ferroptosis" @default.
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