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- W3081789544 endingPage "1117" @default.
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- W3081789544 abstract "Wound repair is a highly regulated process that requires the interaction of various cell types. It has been shown that cancers use the mechanisms of wound healing to promote their own growth. Therefore, it is of importance to identify common regulators of wound repair and tumor formation and to unravel their functions and mechanisms of action. An exciting example is activin, which acts on multiple cell types in wounds and tumors, thereby promoting healing, but also scar formation and tumorigenesis. Here, we summarize current knowledge on the role of activin in these processes and highlight the therapeutic potential of activin or activin antagonists for the treatment of impaired healing or excessive scarring and cancer, respectively." @default.
- W3081789544 created "2020-09-08" @default.
- W3081789544 creator A5014658271 @default.
- W3081789544 creator A5045123741 @default.
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- W3081789544 date "2020-12-01" @default.
- W3081789544 modified "2023-09-25" @default.
- W3081789544 title "Wound Repair, Scar Formation, and Cancer: Converging on Activin" @default.
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- W3081789544 doi "https://doi.org/10.1016/j.molmed.2020.07.009" @default.
- W3081789544 hasPubMedId "https://pubmed.ncbi.nlm.nih.gov/32878730" @default.
- W3081789544 hasPublicationYear "2020" @default.
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